山东大学学报 (医学版) ›› 2018, Vol. 56 ›› Issue (11): 105-110.doi: 10.6040/j.issn.1671-7554.0.2017.1212
• • 上一篇
崔亮亮1,牛纪伦2,于坤坤1,戴志翔2,阮师漫1
CUI Liangliang1, NIU Jilun2, YU Kunkun1, DAI Zhixiang2, RUAN Shiman1
摘要: 目的 定量分析济南市大气污染物SO2和NO2对居民心血管疾病应急呼叫事件的急性影响。 方法 描述性分析2013~2015年济南市逐日气象、空气污染物和居民心血管疾病应急呼叫事件资料。采用基于Quasi-Poisson回归的广义相加模型分别进行SO2、NO2与逐日居民心血管疾病应急呼叫事件的单污染物模型、滞后效应(lag1~lag7)和累积滞后效应(lag01~lag07)分析。计算污染物浓度每升高10 μg/m3,居民心血管疾病应急呼叫事件风险增加的超额危险度及95%置信区间(95%CI)。 结果 2013~2015年济南市逐日SO2、NO2浓度水平及居民心血管疾病应急呼叫事件数的均值分别为73 μg/m3、55 μg/m3及17人。SO2、NO2浓度每升高10 μg/m3,当日(lag0)居民心血管疾病应急呼叫事件风险可分别增加0.41%(95%CI:0.10%~0.72%)和0.99%(95%CI:0.27%~1.71%),同时存在显著的累积滞后效应。 结论 济南市大气污染物SO2、NO2均对居民心血管疾病应急呼叫事件风险存在急性效应。
中图分类号:
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