Journal of Shandong University (Health Sciences) ›› 2024, Vol. 62 ›› Issue (8): 67-73.doi: 10.6040/j.issn.1671-7554.0.2024.0563
DAI Chenyang, GUO Hui
CLC Number:
| [1] Ghenciu LA, Faur AC, Bolintineanu SL, et al. Recent advances in diagnosis and treatment approaches in fungal keratitis: a narrative review [J]. Microorganisms, 2024, 12(1):161. [2] 王晓燕,齐眉,李玉瑭,等.大蒜素与两性霉素B体外联合应用抑制白色念珠菌菌丝形成的研究[J].山东大学学报(医学版), 2010, 48(5): 23-27. WANG Xiaoyan, QI Mei, LI Yutang, et al. Study of the combination of allicin and amphotericin B inhibiting hyphae formation of Candida albicans in vitro[J]. Journal of Shandong University(Health Sciences), 2010, 48(5): 23-27. [3] Das S, Dsouza S, Gorimanipalli B, et al. ocular surface infection mediated molecular stress responses: a review [J]. Int J Mol Sci, 2022, 23(6): 3111. [4] Fang X, Lian H, Bi S, et al. Roles of pattern recognition receptors in response to fungal keratitis [J]. Life Sci, 2022, 307: 120881. doi:10.1016/j.lfs.2022.120881. [5] Pearlman E, Sun Y, Roy S, et al. Host defense at the ocular surface [J]. Int Rev Immunol, 2013, 32(1): 4-18. [6] 李妍,李丽红,戴敏,等.真菌性角膜炎角膜组织NOD1、NF-κBp65表达的变化以及炎症细胞浸润情况[J].眼科新进展, 2023, 43(2): 89-93. LI Yan, LI Lihong, DAI Min, et al. Changes in expression of nucleotide-binding oligomerization domain-contai-ning protein 1 and nuclear factor kappa-Bp65 and infiltration of inflammatory cells in the cornea with fungal keratitis[J]. Recent Advances in Ophthalmology, 2023, 43(2): 89-93. [7] Dai C, Wu J, Chen C, et al. Interactions of thymic stromal lymphopoietin with TLR2 and TLR4 regulate anti-fungal innate immunity in Aspergillus fumigatus-induced corneal infection [J]. Exp Eye Res, 2019, 182: 19-29.doi:10.1016/j.exer.2019.02.020. [8] Han F, Guo H, Wang L, et al. TSLP produced by aspergillus fumigatus-stimulated DCs promotes a Th17 response through the JAK/STAT signaling pathway in fungal keratitis [J]. Invest Ophthalmol Vis Sci, 2020, 61(14): 24. [9] Ren X, Wang L, Wu X. A potential link between TSLP/TSLPR/STAT5 and TLR2/MyD88/NFκB-p65 in human corneal epithelial cells for Aspergillus fumigatus tolerance [J]. Mol Immunol, 2016, 71: 98-106. doi:10.1016/j.molimm.2015.12.014. [10] Xu X, Wei Y, Pang J, et al. time-course transcriptomic analysis reveals the crucial roles of PANoptosis in fungal keratitis [J]. Invest Ophthalmol Vis Sci, 2023, 64(3): 6. [11] Hu J, Hu Y, Chen S, et al. Role of activated macrophages in experimental Fusarium solani keratitis [J]. Exp Eye Res, 2014, 129: 57-65. doi:10.1016/j.exer.2014.10.014. [12] 朱彬彬.NETs的形成对铜绿假单胞菌性角膜炎进展和预后影响的研究[D].杭州: 浙江大学, 2018. [13] Hua X, Chi W, Su L, et al. ROS-induced Oxidative Injury involved in Pathogenesis of Fungal Keratitis via p38 MAPK Activation [J]. Sci Rep, 2017, 7(1): 10421. [14] Bassoy EY, Towne JE, Gabay C. Regulation and function of interleukin-36 cytokines [J]. Immunol Rev, 2018, 281(1): 169-178. [15] Bensen JT, Dawson PA, Mychaleckyj JC, et al. Identification of a novel human cytokine gene in the interleukin gene cluster on chromosome 2q12-14 [J]. J Interf Cytokine Res, 2001, 21(11): 899-904. [16] Dinarello CA. Overview of the IL-1 family in innate inflammation and acquired immunity [J]. Immunol Rev, 2018, 281(1): 8-27. [17] Nicklin MJH, Barton JL, Nguyen M, et al. A sequence-based map of the nine genes of the human interleukin-1 cluster [J]. Genomics, 2002, 79(5): 718-725. [18] Zhou L, Todorovic V. Interleukin-36: structure, signaling and function [J]. Adv Exp Med Biol, 2021, 21: 191-210. doi:10.1007/5584_2020_488. [19] Afonina IS, Müller C, Martin SJ, et al. Proteolytic processing of interleukin-1 family cytokines: variations on a common theme [J]. Immunity, 2015, 42(6): 991-1004. [20] Clancy DM, Henry CM, Sullivan GP, et al. Neutrophil extracellular traps can serve as platforms for processing and activation of IL-1 family cytokines [J]. FEBS J, 2017, 284(11): 1712-1725. [21] Henry CM, Sullivan GP, Clancy DM, et al. Neutrophil-derived proteases escalate inflammation through activation of IL-36 family cytokines [J]. Cell Rep, 2016, 14(4): 708-722. [22] Oneill LA. Towards an understanding of the signal transduction pathways for interleukin 1 [J]. Biochim Biophys Acta, 1995, 1266(1): 31-44. [23] Oneill LA, Greene C. Signal transduction pathways activated by the IL-1 receptor family: ancient signaling machinery in mammals, insects, and plants [J]. J Leukoc Biol, 1998, 63(6): 650-657. [24] Xu Y, Tao X, Shen B, et al. Structural basis for signal transduction by the Toll/interleukin-1 receptor domains [J]. Nature, 2000, 408: 111-115.doi:10.1038/35040600. [25] Sims JE, March CJ, Cosman D, et al. cDNA expression cloning of the IL-1 receptor, a member of the immunoglobulin superfamily [J]. Science, 1988, 241(4865): 585-589. [26] Wang DL, Zhang SY, Li L, et al. Structural insights into the assembly and activation of IL-1β with its receptors [J]. Nat Immunol, 2010, 11: 905-911. doi:10.1038/ni.1925. [27] Medzhitov R, Preston-Hurlburt P, Kopp E, et al. MyD88 is an adaptor protein in the hToll/IL-1 receptor family signaling pathways [J]. Mol Cell, 1998, 2(2): 253-258. [28] Dunne A, ONeill LA. The interleukin-1 receptor/Toll-like receptor superfamily: signal transduction during inflammation and host defense [J]. Sci STKE, 2003, 2003(171): re3. doi:10.1126/stke.2003.171.re3. [29] Tiwari RL, Singh V, Singh A, et al. IL-1R-associated kinase-1 mediates protein kinase Cδ-induced IL-1β production in monocytes [J]. J Immunol, 2011, 187(5): 2632-2645. [30] 杨薇,钟波.IL-36细胞因子在炎性疾病与肿瘤中的作用机制[J].中国细胞生物学学报, 2022, 44(4): 737-746. YANG Wei, ZHONG Bo. The roles of IL-36 cytokines in inflammatory diseases and cancers[J]. Chinese Journal of Cell Biology, 2022, 44(4): 737-746. [31] Yuan ZC, Xu WD, Liu XY, et al. Biology of IL-36 signaling and its role in systemic inflammatory diseases[J]. Front Immunol, 2019, 10: 2532. doi:10.3389/fimmu.2019.02532. [32] Macleod T, Berekmeri A, Bridgewood C, et al. The immunological impact of IL-1 family cytokines on the epidermal barrier[J]. Front Immunol, 2021, 12: 808012. doi:10.3389/fimmu.2021.808012. [33] Russell SE, Horan RM, Stefanska AM, et al. IL-36α expression is elevated in ulcerative colitis and promotes colonic inflammation [J]. Mucosal Immunol, 2016, 9(5): 1193-204. [34] Scheibe K, Backert I, Wirtz S, et al. IL-36R signalling activates intestinal epithelial cells and fibroblasts and promotes mucosal healing in vivo [J]. Gut, 2017, 66(5): 823-838. [35] Queen D, Ediriweera C, Liu L. Function and regulation of IL-36 signaling in inflammatory diseases and cancer development[J]. Front Cell Dev Biol, 2019, 7: 317. doi:10.3389/fcell.2019.00317. [36] Vigne S, Palmer G, Martin P, et al. IL-36 signaling amplifies Th1 responses by enhancing proliferation and Th1 polarization of naive CD4+ T cells [J]. Blood, 2012, 120(17): 3478-3487. [37] Vigne S, Palmer G, Lamacchia C, et al. IL-36R ligands are potent regulators of dendritic and T cells [J]. Blood, 2011, 118(22): 5813-5823. [38] Foster AM, Baliwag J, Chen C S, et al. IL-36 promotes myeloid cell infiltration, activation, and inflammatory activity in skin [J]. J Immunol, 2014, 192(12): 6053-6061. [39] Keller J, O Siorain JR, Kündig TM, et al. Molecular aspects of Interleukin-36 cytokine activation and regulation [J]. Biochem Soc Trans, 2024, 52(4): 1591-1604. [40] Macleod T, Ainscough JS, Hesse C, et al. The proinflammatory cytokine il-36γ is a global discriminator of harmless microbes and invasive pathogens within epithelial tissues [J]. Cell Rep, 2020, 33(11): 108515. [41] Heath JE, Scholz GM, Veith PD, et al. IL-36γ regulates mediators of tissue homeostasis in epithelial cells [J]. Cytokine, 2019, 119: 24-31.doi:10.1016/j.cyto.2019.02.012. [42] Nanjo Y, Newstead MW, Aoyagi T, et al. Overlapping roles for interleukin-36 cytokines in protective host defense against murine Legionella pneumophila pneumonia[J]. Infect Immun, 2019, 87(1): e00583-e00518. [43] Kovach MA, Singer B, Martinez-Colon G, et al. IL-36γ is a crucial proximal component of protective type-1-mediated lung mucosal immunity in Gram-positive and-negative bacterial pneumonia [J]. Mucosal Immunol, 2017, 10(5): 1320-1334. [44] Verma AH, Zafar H, Ponde NO, et al. IL-36 and IL-1/IL-17 drive immunity to oral candidiasis via parallel mechanisms[J]. J Immunol, 2018, 201(2): 627-634. [45] Braegelmann J, Braegelmann C, Bieber T, et al. Candida induces the expression of IL-36γ in human keratinocytes: implications for a pathogen-driven exacerbation of psoriasis? [J]. J Eur Acad Dermatol Venereol, 2018, 32(11): 403-406. [46] Miyachi H, Wakabayashi S, Sugihira T, et al. Keratinocyte IL-36 receptor/MyD88 signaling mediates Malassezia-induced IL-17–dependent skin inflammation[J]. J Infect Dis, 2021, 223(10): 1753-1765. [47] Gao N, Me R, Dai C, et al. Opposing effects of IL-1Ra and IL-36Ra on innate immune response to Pseudomonas aeruginosa infection in C57BL/6 mouse corneas[J]. J Immunol, 2018, 201(2): 688-699. [48] Dai C, Me R, Gao N, et al. Role of IL-36γ/IL-36R signaling in corneal innate defense against Candida albicans keratitis[J]. Invest Ophthalmol Vis Sci, 2021, 62(6): 10. [49] Cho WJ, Elbasiony E, Singh A, et al. IL-36γ augments ocular angiogenesis by promoting the vascular endothelial growth factor-vascular endothelial growth factor receptor axis[J]. Am J Pathol, 2023, 193(11): 1740-1749. [50] Chen Q, Gao N, Yu FS. Interleukin-36 receptor signaling attenuates epithelial wound healing in C57BL/6 mouse corneas[J]. Cells, 2023, 12(12): 1587. [51] Kuo MT, Chen JL, Hsu SL, et al. An omics approach to diagnosing or investigating fungal keratitis[J]. Int J Mol Sci, 2019, 20(15): E3631. [52] Sharma N, Bagga B, Singhal D, et al. Fungal keratitis: a review of clinical presentations, treatment strategies and outcomes[J]. Ocul Surf, 2022, 24: 22-30. doi:10.1016/j.jtos.2021.12.001. [53] Zemba M, Radu M, Istrate S, et al. Intrastromal injections in the management of infectious keratitis[J]. Pharmaceutics, 2023, 15(4): 1091. [54] Sha XY, Shi Q, Liu L, et al. Update on the management of fungal keratitis[J]. Int Ophthalmol, 2021, 41(9): 3249-3256. [55] Zou HY, Wang HT, Xu BQ, et al. Regenerative cerium oxide nanozymes alleviate oxidative stress for efficient dry eye disease treatment[J]. Regen Biomater, 2022, 9: rbac070. doi:10.1093/rb/rbac070. [56] Niu PH, Wu YL, Zeng FX, et al. Development of nanodrug-based eye drops with good penetration properties and ROS responsiveness for controllable release to treat fungal keratitis[J]. NPG Asia Mater, 2023, 15: 31. doi:10.1038/s41427-023-00478-9. [57] Chen HY, Geng XW, Ning QY, et al. Biophilic positive carbon dot exerts antifungal activity and augments corneal permeation for fungal keratitis[J]. Nano Lett, 2024, 24(13): 4044-4053. [58] 徐霁,周文君,蔡春华,等.聚乙二醇化壳聚糖纳米粒的制备及局部滴眼给药性能评价[J].第三军医大学学报, 2017, 39(13): 1376-1380. XU Ji, ZHOU Wenjun, CAI Chunhua, et al. Preparation and performance of PEGylated chitosan nanoparticles for ocular drug delivery by eye drops[J]. Journal of Third Military Medical University, 2017, 39(13): 1376-1380. [59] Wong KY, Wong MS, Liu J. Nanozymes for treating ocular diseases[J]. Adv Healthc Mater, 2024: e2401309. doi:10.1002/adhm.202401309. |
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